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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. pennis enlargement surgery photo penis enhancement vimax penis enlargement surgery cost natural penis enhancement pills penile enlargement result penis elargement tool free pnis enlargement technique free exercise tip for penis enlargment

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There are nonsurgical options for prostate cancer. Hormonal therapy is just as popular as surgery, but maybe dismissed by many urologists as unproven. Many patients whose cancer has spread throughout the body find relief through reducing the amount of male hormone in the bloodstream which is what the cancer “feeds on". Without testosterone, impotency is almost inevitable. One such drug is the DES (diethistibestrol), a chemical that is similar to estrogen (a female hormone). Although DES brings with it a longer life and lessens the symptoms, it does have side effects. It kills a man’s sex drive, and many men report the nearly female effects: breast enlargement, thinning beards and even scrotal shrinkage. LH-RH Agonists (Leuprolide and Goserelin) can be used in later stages of prostate cancer. This is a synthetic pituitary hormone that regulates the release of testosterone into the bloodstream. After triggering an initial rush of testosterone, it causes a drastic decrease in testosterone which then starves the tumor. The side effects are occasional hot flashes, headaches, or impotence. It is usually administered through injections or implant pellets. Antiandrogens are hormones that inhibits the action of testosterone on cancer cells. It is also marketed to the brand name Flutamide, and it is frequently used in conjunction with other hormone treatments to lessen the pain of advanced prostate cancer Immunotherapy is a method used to build up the body's natural defense against diseases, including cancer. Some doctors combine immunotherapy with chemotherapy to help patients through the later stages of prostate cancer. Other sufferers use natural cures and herbs to help their bodies fight the cancer. Radiation and Hyperthermia Scientists do know that temperatures of 105°F or higher are deadly to cell division and weak cells. Scientists are attempting to put this information to work on prostate cancer by combining it with radiation to kill cancer cells. When applying heat to the whole body or to the localized area prior to radiation, it seem s to be more effective than either treatment alone There are Enzyme-inhibiting drugs such as Proscar by blocking production of an enzyme that triggers production of testosterone. Chemotherapy is usually considered a last resort for many cancer patients. The chemicals directly attack the cancer cells but usually end up killing many healthy cells as well. These are only a few of the treatments available for prostate cancer patients. Please discuss with your doctor as to what treatment may be the best option for you. best penis enlargement penis enlarement surgery picture vimax penis enlargement secret penile enlargment surgery pnis enlargement device best penis enargement vimax do penis enlargement pills really work cheap penis enlargment pills penis enlarement surgery picture

The penis enlargement market is growing by the day as more and more men join the trend that’s slowly moving into the mainstream. Men worried about the size of their penises and men who are doing just fine, but want to gain a little extra length, are all starting on penis enlargement. But the question is: how many of them actually manage to successfully finish their enlargement programs? And what are the causes of failures reported by these men. Well, the top reasons why men fail in their quest for bigger penises are boredom and lack of motivation. You have to realize that many men fail simply because they aren’t motivated enough. They are enthusiastic about starting their penis enlargement exercises, but lose interest after some time. Some don’t have the willpower to stick to the program and get depressed over the lack of results. Others are simply bored by everything that doesn’t provide results within five minutes. Still others decide that penis enlargement is too much trouble. Another big issue, one that plagues committed penis enlargement customers, is the incorrect assessment of workout intensity. In time, some users find a certain performance pace in their workouts and fail to realize that workout intensity should increase over time. The intensity of a beginner’s workout is no longer suitable after two months of workouts. And at the opposite pole are the guys who rush straight into a high-intensity workout without going through the beginner phase. These people always manage to injure themselves by trying to cheat time and obtain results way ahead of schedule. Both these groups end up in the demotivation zone simply because they are out of sync with the proper workout schedule. In a similar situation are those who aren’t aware that they should change their routines. As the body adapts to the exertion, heavily used exercises become less and less effective and should be replaced with new ones. There are plenty to choose from, so that shouldn’t be a problem. Keep in mind that once you’ve squeezed everything from an exercise, it’s time to move on to other routines. It’s the only way those inches will start adding. Not paying proper attention to advice offered by penis enlargement experts is also a big problem. For instance, men who engage in penis enlargement exercises are told time and again that they should abstain from sex for two or three hours after finishing a workout session. The penis needs time to heal and rebuild broken tissues and further exertion is likely to interfere with this process. Still, some people refuse to follow this piece of advice. While I do understand that it’s a shame to waste a perfectly good hard-on, I also know that advice coming from experts should be heeded. Not paying proper attention to exercise instructions is even worse of a problem. Although the exercises are fairly easy to follow, especially with a video demonstration attached, some people fail to get them right. This is the reason behind most of the injuries reported by penis enlargement users. People failing to perform exercises as shown or trying to improve on the design. There are also people who don’t know when to stop and think they can tank through the pain and make it on the other side through sheer willpower. Although a certain level of pain is normal during penis enlargement exercises, intense pain is a clear sign that you should stop whatever it is you’re doing and let your penis recover. And the last issue on my list of mistakes is warming up and down. This is yet another thing that men are told they must do before and after workout sessions. Still, some men choose to skip this part of the program. Warming up and down is very important to preparing the tissues for the coming workout and for the healing and rebuilding process. Although some men can do without the warming up and down, most men should follow these procedures. It is in their best interest. So there you have it. A list of common mistakes people make and which hinder their efforts to add those extra inches to their penises. Forewarned, you can avoid these pitfalls and keep on the path to safe and effective penis enlargement. Have fun and stay safe. vigrx penis enlargement pills best elargement exercise penis vimax testimonials safe penile enlargment does penile enlargement work vimax penis enlargement drug penis enhancement exercise herbal penis enlarement pills penis enlarement surgery picture

Generally, what we understand when we first hear about Cialis is the fact that this is the first true competitor of Viagra as it fights against erectile disfunction as well as the first mentioned. Viagra appeared in 1998 and registered a high success generally for the fact that it was the first product of this kind. Nowadays a new successful product was attributed to ICOS corporation which is to shatter the unbelievable success of Viagra. The series isn't ending here, anyway,as another one called Levitra is expected to appear. First of all, there should be mentioned that the sell market of Cialis has reached an enormous level which is bound to put aside the success registered by Viagra in the late 90's. What is important is the fact that from around thirty million people suffering from erectile disfunction in the USA and another estimated number of one hundred and seventy five million abroad who are having this disfunction and using these products, only a little part are using Viagra. Viagra has reached this year a total profit of $1,5bn, while the account of money used for these drugs(such as Viagra, Levitra and Cialis) raises up to $6. Similar to the case of Viagra, Cialis should be understood as a general performance provider, to just a regular treatment for the ailment. Anyone of us should know that there is a specific process of these medication which makes them be so successful, while constantly improving our lives: they work by blocking an enzyme named phosphodiesterase (which happens to relax some of our muscles), this way allowing a certain growth of the blood flow in your penis. Another advantage gained by Cialis over Viagra is the fact that it is available(by prescription only) in countries like the United Kingdom, Denmark, Finland, Sweden and Australia and it is ought to be approved in USA too. Moreover, a common argument brought in favor of Cialis is the fact that it has more rapid effects than Viagra; The starting process can be reached at approximately sixteen minutes after taking the pill, while the entire process can lead up to twenty four hours or more. In contrast to that, by taking Viagra one may feel the effects after one hour and have them for four hours upmost. Like Viagra, an estimated number of eighty percent of the people who have tried Cialis have had the opportunity and chance in the mean time to have an erection as well as sexual acts but in comparison with Viagra, the effects and potential of the user lasted for more than thirty six hours after taking the pill;in this period of time the effects were almost constant. A possible explanation for this is the fact that Cialis persists longer in the body than Viagra does. The period of time for blood levels to fall fifty percent for Viagra records four hours, while Cialis makes it up to seventeen hours. This can signify that a twenty five percent of the original doze of Cialis is still in the body at that time. To conclude, it may be said that Cialis has finally shattered the’ unbreakable’ myth of Viagra by adding more tough points to the well-known advantages of Viagra. Logically, it is of a higher quality technique than Viagra this being the reason for the extreme surpassing. Let’s hope things will not remain this way and will improve more and more up to perfection semenax vig rx buy penis enlargement pill penis enlarement without pills vimax top rated penis enlargement pills penile enlargment exercise vimax guide to penis enlargement vimax enlargement free penis pills sample buy place vigrx penis enlarement surgery picture

Gonorrhea is a STD. it is also called clap. Bacteria spread gonorrhea. Most of us believe that kissing is very harmless. But kissing can cause Gonorrhea. Let me tell you more. Gonorrhea - the affected areas Gonorrhea bacteria affect most of the places in the body with mucous membrane. That includes the genitals, the anus and rectum, throat, and possibly eyes. The bacteria pass from secretions of any of the affected areas. Once your mucous membrane comes in contact with secretions of an infected person, you may contact Gonorrhea. Any sexual activity will transfer the bacteria. if the bacteria has invaded the throat of the infected person, a kiss will transfer it. If such a person performs oral sex on you, you will get Gonorrhea. Gonorrhea complications- Gonorrhea is a disease, which is better avoided. if it is left untreated, it can cause infertility in both men and women. In men it may even close the urethra( from where men pass the urine) , infect the testicles and create other complications, In women it may infect the fallopian tubes and may cause PID- Pelvic Inflammatory Disease. Gonorrhea - the early symptoms Yellowish discharge from the penis or vagina, painful passing of stools, burning sensation in the genitals are some of the common symptoms of Gonorrhea. To know more about Gonorrhea please click here- Gonorrhea This article is only for informative purposes. This article is not intended to be a medical advise and it is not a substitute for professional medical advice. Please consult your doctor for your medical concerns. Please follow any tip given in this article only after consulting your doctor. The author is not liable for any outcome or damage resulting from information obtained from this article.